{
  "title": "What this review means",
  "includeSubclaimIllustrations": false,
  "redteamReviewed": true,
  "accessNotice": "Source checking used bounded original primary abstract passages/status records and selected original partial-body passages. No complete-abstract or complete-paper audit, or independent scientific peer review, is claimed. Detailed access counts and limitations appear in the review.",
  "overview": "Dopamine dysregulation is relevant to psychosis and symptom modulation; unselected mesolimbic D2 primacy is not established.",
  "implications": "Overall Γ follows registered SC3, with association SC1 at Β and a conservative Γ for the strictly audited SC2 mechanism gate. The nominal claim fixes mesolimbic anatomy and unselected primacy, whereas strong human findings often concern associative striatum or symptom-responsive contexts. Clinical symptom modulation remains supported; the unresolved exact causal-primacy claim cannot be rescued by changing the anatomical label or retrospectively selecting responders.",
  "limitations": "50 primary reports with 3 selected partial-body examinations; density, heterogeneous controls and incomplete access set the confidence, not correctness. The grade applies to the exact registered claim; stronger findings for narrower subclaims remain separate. No complete-paper audit or blinded calibration is claimed. This educational review does not establish an individual treatment recommendation."
}