{
  "completed": true,
  "status": "documented_historical_discovery_and_reconciliation",
  "provider": "Consensus",
  "provider_mode": "Historical browser/connector use as recorded in the source artifacts",
  "local_audit_date": "2026-10-09",
  "primary_comparison_count": null,
  "reported_usage": [
    {
      "path": "D:\\HypothesisCritic\\deliverables\\hsef_v53_popularity_ranks_41_45_2026-10-08\\microbiome-causality\\consensus\\usage.json",
      "metrics": {
        "deep_queries": 0,
        "api_calls": 0,
        "browser_pro_queries": 2,
        "targeted_studies_requested": 9,
        "original_passage_comparisons": 4
      }
    }
  ],
  "grade_changed": false,
  "score_before": {
    "hsef_level": "Γ",
    "hsef_descriptor": "Mechanistically plausible, partial system support",
    "confidence": "Moderate",
    "confidence_rationale": "67 primary reports with 8 selected partial-body examinations; density, heterogeneous controls and incomplete access set the confidence, not correctness.",
    "rests_on_non_peer_reviewed_work": false,
    "rationale": "SC4 is the preregistered human-primacy gate. Transfers, defined strains and microbial products show real bounded contributions, and HE intervention crosses human contribution thresholds, but none establishes composition as the primary upstream target across systemic and neurobehavioural disease. SC1 association is Β, bounded rodent causality SC2 Ζ, human contribution SC3 Ε, broad primacy SC4 Γ. These are different scopes, not a refutation of the microbiome."
  },
  "score_after": {
    "hsef_level": "Γ",
    "hsef_descriptor": "Mechanistically plausible, partial system support",
    "confidence": "Moderate",
    "confidence_rationale": "67 primary reports with 8 selected partial-body examinations; density, heterogeneous controls and incomplete access set the confidence, not correctness.",
    "rests_on_non_peer_reviewed_work": false,
    "rationale": "SC4 is the preregistered human-primacy gate. Transfers, defined strains and microbial products show real bounded contributions, and HE intervention crosses human contribution thresholds, but none establishes composition as the primary upstream target across systemic and neurobehavioural disease. SC1 association is Β, bounded rodent causality SC2 Ζ, human contribution SC3 Ε, broad primacy SC4 Γ. These are different scopes, not a refutation of the microbiome."
  },
  "independent_blinded_audit": false,
  "raw_receipts": [
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_41_45_2026-10-08\\microbiome-causality\\consensus\\broad-ax.txt",
      "sha256": "4eb87152f7a5f5d3242b2967432f9786fefe1f7bb85c7efc96bf6242bfa30d21"
    },
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_41_45_2026-10-08\\microbiome-causality\\consensus\\reconciliation.md",
      "sha256": "6a015788c3d0afdf44f153c5ba16534e686763809a191e970d41fc53707d83c6"
    },
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_41_45_2026-10-08\\microbiome-causality\\consensus\\targeted-ax.txt",
      "sha256": "804f5fdf3c37137d53b64fda1918947c17cefed00d8dcf840897e05d5a0b7206"
    },
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_41_45_2026-10-08\\microbiome-causality\\consensus\\usage.json",
      "sha256": "e041f8084a12ffe6d8a4bcdadea42c0044b3739845d8e88e110d82f00867cfb7"
    }
  ],
  "limitations": "This verifies documented historical use and links the record to the final edition. It adds no primary-source, full-text, independence or verification credit. Original access remains limited to the source-specific record."
}