{
  "completed": true,
  "status": "documented_historical_discovery_and_reconciliation",
  "provider": "Consensus",
  "provider_mode": "Historical browser/connector use as recorded in the source artifacts",
  "local_audit_date": "2026-10-09",
  "primary_comparison_count": null,
  "reported_usage": [],
  "grade_changed": false,
  "score_before": {
    "hsef_level": "Γ",
    "hsef_descriptor": "Mechanistically plausible, partial system support",
    "confidence": "Moderate",
    "confidence_rationale": "69 primary reports with 6 selected partial-body examinations; density, heterogeneous controls and incomplete access set the confidence, not correctness.",
    "rests_on_non_peer_reviewed_work": false,
    "rationale": "The registered weakest-link conjunction returns Γ because disease-class human generalization remains unresolved. Defined glial states have real causal effects on neuronal loss and progression, including reactive amplifiers. This evidence is not dismissed for being downstream. Opposite effects of protective immune programs and negative or harmful human interventions prevent treating chronic activation as one broadly pathogenic exposure. The limiting gate is generalization and state-specific human mediation, not an added obligation to initiate disease."
  },
  "score_after": {
    "hsef_level": "Γ",
    "hsef_descriptor": "Mechanistically plausible, partial system support",
    "confidence": "Moderate",
    "confidence_rationale": "69 primary reports with 6 selected partial-body examinations; density, heterogeneous controls and incomplete access set the confidence, not correctness.",
    "rests_on_non_peer_reviewed_work": false,
    "rationale": "The registered weakest-link conjunction returns Γ because disease-class human generalization remains unresolved. Defined glial states have real causal effects on neuronal loss and progression, including reactive amplifiers. This evidence is not dismissed for being downstream. Opposite effects of protective immune programs and negative or harmful human interventions prevent treating chronic activation as one broadly pathogenic exposure. The limiting gate is generalization and state-specific human mediation, not an added obligation to initiate disease."
  },
  "independent_blinded_audit": false,
  "raw_receipts": [
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_21_25_2026-10-06\\neuroinflammation-ranked-refresh\\consensus\\broad-browser-snapshot.txt",
      "sha256": "a6fe2688372703af971bd35ce5aa7b59e66171cceb05215d5ddc4251481dd0ff"
    },
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_21_25_2026-10-06\\neuroinflammation-ranked-refresh\\consensus\\query-ledger.json",
      "sha256": "6f48a021f249a5f2d15e4d7eb04bcf45c5e38e56d80dc5c9b8c15bdb5dbfa240"
    },
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_21_25_2026-10-06\\neuroinflammation-ranked-refresh\\consensus\\reconciliation.md",
      "sha256": "b3a7ea6a5c74184bab9f6ccc5e5bce46fb22c8a82a380644af02f4579157ebb6"
    },
    {
      "path": "deliverables\\hsef_v53_popularity_ranks_21_25_2026-10-06\\neuroinflammation-ranked-refresh\\consensus\\targeted-browser-snapshot.txt",
      "sha256": "0ebe1836b56e09d1986def71edc5697bf12e7cc0bb5b76b590ef0a424a5412f4"
    }
  ],
  "limitations": "This verifies documented historical use and links the record to the final edition. It adds no primary-source, full-text, independence or verification credit. Original access remains limited to the source-specific record."
}